
Shifts in commensal microbiota composition are emerging as a hallmark of gastrointestinal inflammation. In particular, outgrowth of γ-proteobacteria has been linked to the etiology of inflammatory bowel disease and the pathologic consequences of infections. Here we show that following acute Toxoplasma gondiigastrointestinal infection of mice, control of commensal outgrowth is a highly coordinated process involving both the host response and microbial signals.
Learn more